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Intravenous infusion of aldosterone (10 µg/kg body wt per h for 5 h) in four 2-month-old calves decreased salivary and urinary sodium (Na⁺) concentration and increased salivary potassium (K⁺) concentration without modifying salivary flow or urinary K⁺ concentration. Intravenous angiotensin II infusion (0·3 µg/kg body wt per min for 1 h) in four Na⁺-replete 16-month-old bulls decreased salivary Na⁺ concentration and increased that of K⁺. It also increased plasma cortisol and plasma aldosterone concentrations, and decreased plasma renin activity (PRA). In four 16-month-old bulls Na⁺ deficiency (induced by chronic cannulation of the righ parotid duct and loss of saliva for 5 days) had similar effects to those observed following aldosterone infusion in calves: a decrease in salivary Na⁺/K⁺ ratio. This decrease was associated with an increase in PRA and an increase in plasma aldosterone concentration. In these animals a close positive relationship was observed between PRA and plasma aldosterone concentration (r = 0·91; n = 20; P < 0·01). Thus in cattle, during Na⁺ deficiency, the effect of aldosterone on parotid glands participates in the regulation of Na⁺ metabolism.

J. Endocr. (1986) 108, 405–411