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Specific inhibition of glutamic acid decarboxylase (GAD, EC 4.1.1.1 [EC] 5; the main enzyme involved in the synthesis of -aminobutyric acid) by mercaptopropionic acid interferes with the effect of dexamethasone on both the resting and stress-induced secretion of ACTH. It is postulated that dexamethasone may, at least in part, inhibit the secretion of ACTH via the induction of GAD, thereby raising the level of -aminobutyric acid in the central nervous system.

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				L. B. Mercado-Asis, J. A. Yanovski, H. L. Tracer, C. L. Chik, and G. B. Cutler Jr. Acute Effects of Bromocriptine, Cyproheptadine, and Valproic Acid on Plasma Adrenocorticotropin Secretion in Nelson's Syndrome J. Clin. Endocrinol. Metab., February 1, 1997; 82(2): 514 - 517. [Abstract] [Full Text] [PDF]